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Illumination of the kitchen
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Index ,
Connections ,
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Info Patient
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SYNDROMES OF THE MOTOR
Differentiated Diagnosis
Hereditary
Sclerosis of lateral of Amyotrophic
Hereditary
Sporadic
Variants of ALS
EOM and extrapiramidal change
Primary lateral sclerosis
Primary muscular Atrophy
Western Pacific ALS
Amyotrophy
Lower the extremity, benign
Monomelic
Diabetic Amyotrophy
Weakness of the hand
Hopkins
Infections
Insulinoma
Mitochondrial: SCO2
Disorders Of the Neuron Of the Motor
Hereditary
Bulbar
Lower the neuron of the motor
Superior neuron of the motor
Asymmetric
Symmetrical
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Neuropatía Of The Motor
Distal
IgM against ganglioside GM1
IgM against ganglioside of GalNAc-GD1a
Multifocal Neuropatía of the motor (MMN)
Myopathies
Painful
Paraneoplastic
Linfoma
Chest
Syndrome of the poliomielitis and Poste-poliomielitis
Fast beginning
Acute Neuropatía Of The Motor De Axonal
Poliomielitis
Porphyria
Spinal muscular Atrophy: Hereditary
SMN (5q)
Receiver Of The Androgen (SMA Bulbar)
SMA Distal
Hexosaminidase A (Tay-Sachs)
Scapuloperoneal
Spinal muscular Atrophy 2
X-Related Infant SMA And Arthrogryposis
Espástico Paraparesis
Toxic
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Of Bramwell: Atlas of the clinical medicine
ALS with the implication to bulbar
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Disorders Of the Neuron Of the Motor:
Diagnosis Differentiated
- Asymmetric
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- Stop lower + Muestras Of the Neuron Of the Motor
- Sclerosis De Lateral De Amyotrophic (ALS)
- Sporadic ALS
- Hereditary ALS
- Dismutase De Superoxide; Chromosome 21;
Dominant
- The Other ALS: Recesivo And Dominante
- Sporadic disorders of the neuron of the motor with
dementia
- Western Pacific ALS
- The frontal dementia followed by disease of the
system of the motor
- Superior neuron of the motor: Specially to
bulbar
- Lower the neuron of the motor:
Fasciculaciones; Smooth weakness
- Abnormal syndromes of Creutzfeld-Jacob
- Disorders of the multi-system
- Extrapiramidales ALS with Ophthalmoplegia and
disorders
- Disease of the body of Polyglucosan
- Motor neuronopathy with the skeletal cataracts
and abnormalitys
- Multiple Atrophy of the system
- Syndrome of the AAA
- Only lower the samples of the neuron of the motor
(asymmetric)
- Syndrome lower Distal Of the Neuron Of Motor
(LMN)
- IgM against ganglioside GM1
- IgM against ganglioside of GalNAc-GD1a
- Also it sees: Multifocal Neuropatía of the
motor
- Next lower syndromes Of the Neuron Of the Motor
- Brachial Diplegia amyotrophic
- Associated to IgM against asialo-GM1
- Rare: Stop > lower members with the
antibodies of anti-Hu
- Lower the syndrome of the neuron of the motor
without the antibodies (PMA)
- Variants of ALS
- Focal disease of the neuron of the motor
- Monomelic Amyotrophy
- Muscle of Paraspinous amyotrophy
- Cervical Amyotrophy
- Motor of Paraneoplastic neuro(no)pathy
- Smooth weakness: With linfoma
- Severe weakness: With the chest cancer
- Syndrome of Hopkins: Acute post-asthmatic
Amyotrophy
- Syndrome of the poliomielitis and
Poste-poliomielitis
- SMN2 (canceladurasof SMN C)
- Neurofibromatosis, Type 2 5
- Symmetrical And Next :
Hereditary Spinal Muscular Atrophy
- SMN: Chromosome 5q; Recesivo
- Receiver Of The Androgen (Bulb-Spinal Muscular
Atrophy): X-bound; Recesivo
- Hexosaminidase A (Tay-Sachs): Chromosome
15; Recesivo
- Weakness of the hand
- Neuropathies of the disease and the motor of the
neuron of the motor
- Myopathies: Distal; Dystrophy De
Myotonic; Myositis body Of the Inclusion
- Myasthenia Gravis
- Peripheral injury of the nerve
- Midpoint: Recurrent branch of the motor;
Previous Interosseus
- Cubital: Channel of Guyon
- Radial part: Later Interosseus
- Brachial Plexopathy
- Implication to bulbar
- Bulb-Spinal Muscular Atrophy
- Receiver Of The Androgen: X-bound
- Dominant
- Sclerosis De Lateral De Amyotrophic (ALS)
- Sporadic ALS
- Hereditary ALS
- ALS with beginning to bulbar, benign course and
the bodies of Bunina
- Syndromes to bulbar hereditary
- Laere Brown-Vialetto-Van
- Fazio-Londe
- Syndrome to bulbar congenital
- Syndrome of Worster-Sequi'a: Paralysis to
suprabulbar congenital
- Painful : Diabetic
Amyotrophy
- Fast Beginning
- Acute Neuropatía Of The Motor De Axonal
(with jejuni or the IgG serum of campylobacter against
GM1)
- Poliomielitis
- Porphyria
- 4 types neurological attacks of the cause
- Intermittent acute
- Vary Porphyria
- Coproporphyria
- δ- acid deficiency amino-amino-levulinic of
dehydratase
- It tinkles: All the types produce increased δ- amino-amino-levulinic acid during attacks
- Acquired
- Toxic: Lead;
Dapsone; Botulism; Paralysis Of the Signal
- Infections
- Poliomielitis
- The Nile Of the west
- Central European Encefalitis
- Creutzfeld-Jacob
- Amyotrophy
- Polyneuropathy (± Demyelinating)
- Another one
- SMA and syndromes of the neuron of the motor:
Another hereditary one
- Neuropatía Multifocal Of Motor (MMN)
Neuropatía Of The Motor De Paraneoplastic
- Beginning: After the diagnosis of the
tumor
- Epidemiología: Man of the majority and >
50 years
- Clinical
- Weakness
- Asymmetric; Arms > Legs
- Smooth
- Only lower the neuron of the motor
- Normal Bulbar
- Cramps: Painful
- Without pain in some patients
- Course: The progressive later becomes
stabilized or improves
- Associated a
- Linfoma De Non-Hodgkin
- Also others linfomas and disorders
myeloproliferative 4
- Laboratory
- Csf: No cells; Smoothly increasing
protein
- MRI: Normal spinal marrow
- Neuronopathy
- Model animal: Similar disorder in virus
leukemic murine of the mice 2°
- Also it sees: A Syndrome More under the
Neuron Of the Motor De Paraneoplastic
A lower Syndrome 8 Of the Neuron Of the Motor De
Paraneoplastic
- Epidemiología: Single patient, old female
of 72 years
- Beginning: 4 months before the diagnosis of
the tumor
- Clinical
- Weakness
- Asymmetric in the beginning
- Arms And Legs
- Severe
- Only lower the neuron of the motor
- Bulbar: Hypophonia; Disfagia;
Unilateral face Paresis
- Without pain
- Course
- Excessive months of the progressive
- Improvement after the retirement of the tumor
- Residual incapacity in the long term
- Sensation: Normal
- Reflections of the sinew: Absent
- Cns: Transitory vertigos and nistagma
- Associated a
- Adenocarcinoma de Ductal of the chest
- Laboratory
- Antibodies
- Serum that ties βto spectrin of the
intravenous one, isoform I (Wendish in MW 250kD and 140kD)
- Serm that ties to the segments of the initial of
axon and to the nodes of Ranvier in brain of the rat
- Electrofisiología
- EMG: Denervation
- NCV: Small CMAPs; No block of the
conduction
- MRI: Spinal marrow with the high points of
the signal in t2
- Neuronopathy
- Inmunosupresivo treatment: No answer
- Also it sees: Neuropatía Of The Motor De
Paraneoplastic
HOPKINS SYNDROME: Acute post-asthmatic
Amyotrophy
- Age: 1 to 13 years
- Beginning
- After acute asthmatic attack: Latency 1 to
18 days
- Smooth pain: Member, neck or meningismus
- Fast weakness of the beginning
- Weakness
- Single member; Asymmetric; > can be
next distal
- Severity: Smooth to severe
- Arm or leg
- Sensorial: Normal
- Csf
- Pleocytosis
- Protein: ± Increased
- MRI: Signal of the demonstration of May
(t2) in spinal marrow
- Prognosis: Permanent paralysis
Syndrome Of the Poliomielitis Of the Post
|
Old Poliomielitis Asymmetric Atrophy and weakness
Right leg atrophic (arrow) in patient
with quadriplegic poliomielitis 70 years inside further
on
|
- Criteria Of diagnosis: Clinical
- History of the poliomielitis
- Neurological recovery and functional partisan or
completes
- Stable function > 15 years
- Beginning of
- Fatigue
- Pain the muscle
- Functional loss
- Generally disorder of 2° Musculoskeletal
- The new weakness is rare
- Neurological Examinación
- Lower the syndrome of the neuron of the motor
(confirmed by EMG or MRI)
- The mensurable loss of force is rare
- Reflections diminished or absent of the sinew
- No sensorial loss
- Another one
- No other explanation for the symptoms
- The common pain and the cold intolerancia can
accompany the syndrome
- Characteristics of the laboratory
- CK: Elevated
- Electrofisiología
- Very great units of the motor (up to 10 normal
times)
- Some fibrilaciones and positive acute waves even
in stable patients
- Pathology of the muscle
- Near the complete recovery of the force and the
stable
- Type the predominance of the fiber of the muscle
of I
- Persistent or new weakness
- type fibers of 50% of I
- Angular fibers; Atrophy + hipertrofia
- New high weakness and CK
- Smaller angular fibers
- Necrosis of the fiber of the muscle
- Recommended exercise
- Smooth Paresis: Training of the resistance
- Moderate Paresis: SubMaxima resistance
- Severe Paresis: None; Common
supporting
Monomelic Amyotrophy
- Eponyms
- Disease of Hirayama: Progressive weakness
on 1 to 4 years, then plateau
- Syndrome of ÓSullivan-McLeod: Slow
progression
- Beginning: Young adult; 15 to 25
years; Up to 40 years in India
- Epidemiología
- Man > Female: Until 10:1
- Familiar antecedents
- Generally sporadic
- Occasional familial occurrence
- Field common in the India of east 7
- Weakness
- Prisoner often to a single arm
- Distal implication (97%): C7, C8 and the T1
innervated the muscles; Hand And Forearm
- Next > Distal: 10%
- Side: The Right = Gone
- Atrophy: "amyotrophy oblique"; To
save brachioradialis
- Tremor (80%): In the extension of the
finger; Irregular And Heavy (Minipolymyoclonus)
- Occasional other characteristics
- Weakness
- Shoulder of Ipsilateral
- Progression to the opposite member (18%): A
weakness generally smoother than 1r member
- Worsening in contact with cold
- Fasciculaciones: In the affected side (47%
to 66%); It cannot be symptomatic
- Cramps (16%)
- Sensorial loss: Smooth
- Hiperhidrosis
- Reflections of the sinew: Generally normal
- No craneal nerve, pyramidal leg or changes
- Course of the disease
- Typical syndrome 10 of Hirayama
- Progression: On 1 to 5 years; Once in
a while while 8 years
- Atmospheric disturbances after the phase of the
progression: It can persist per decades
- Incapacity: Smooth or none in 73%
- Syndrome 6 of ÓSullivan-McLeod : Excessive
decades of the progression
- Laboratory
- EMG: Chronic Denervation
- In limb(s) affected (100%)
- Opposite arm or lower extremities in some
patients
- Samples of denervation acute in 45%
- NCV: Small CMAPs in affected members
- Comprehensive answer of the skin: It can be
abnormal
- MRI
- Some patients with hard inelástico:
Compression of the spinal marrow with flexion 1 of the neck
- Another one studies 3
- No important spinal anomalies
- Atrophy of the spinal marrow: C6 and C7
- Signal of t2 in previous horns of the gray
matter
- Flexion-induced breakup smooth of the cord
Amyotrophy: Benign lower extremity 11
- Epidemiología
- 16 described cases
- Man > Female: 5:1
- Beginning: Age mediates 41 years
- Clinical
- Weakness: Later legs
- Generally: Later yearling calf;
Peronei; Sinews of the arched one
- Rare: Quadrangles
- Often bilateral (50%)
- Clinical or subclinical
- Asymmetry: Some
- No sensorial, to bulbar or superior samples of
the neuron of the motor
- Course
- Insidioso beginning
- Slow progression: Few years
- Stabilization
- Laboratory
- MRI
- Loss of muscle with the fat replacement
- Distribution: Lower the extremities
- Distal leg: Later compartment
- Thigh: Long head of the biceps
- EMG: Denervation
- Biopsy of the muscle: Denervation;
Grouped Atrophy
Lower member benign amyotrophy: MRI |
Of: Al-Lozi-Lozi Of M
Legs: Implication of
the later groups of the muscle
|
Of: Al-Lozi-Lozi Of M
Thigh: Long head of
the implication of the biceps
|
"Primary Muscular Atrophy" (PMA)
Gowers
|
Primary muscular Atrophy
Observe to lose, including
muscles paraspinous.
|
- Extensive lower syndrome Of the Neuron Of the Motor
- Weakness: Distribution
- Distal And Pro'ximo: Anyone can be more
prominent
- Asymmetric
- Paraspinous often implies respiratory muscles
and
- It often saves the musculatura to bulbar
- Spontaneous activity of motor
- Cramps: Field common in legs, at night
- Fasciculaciones
- No samples of the neuron of the motor of the
stop
- Pain: Related to the inmovilidad
- Course of the time
- Progressive
- Similar to, faster, or slower than, typical ALS
- Laboratory
- Pathology of the muscle: Atrophy > type
grouped grouping of the fiber
- No antibodies of the serum
- No block of the conduction
- No evidence for the answer to the treatment
- Differentiated Diagnosis
- Next lower syndrome of the neuron of the motor
- ALS
- Sporadic
- Hereditary
- Mutations of the TURF: A4V, Leu84Val,
D101N
- Pathology
- Loss of neurons of the motor in previous horn of
the spinal marrow
- Contraction of the remaining neurons of the
motor
- Bodies of the inclusion:
Intracitoplásmico, Hyaline
Primary Lateral Sclerosis 2
Recesivo, beginning of the childhood
Sporadic, beginning of the adult
|
PLS: Sporadic, beginning of the adult
- Discreet syndrome against ALS variant
- Clinical
- Beginning
- Slowly progressive
- Legs before arms
- The more commonly possible in 5ta decade
- No familiar antecedents
- Disfunción of the motor
- Disfunción ± to corticobulbar of the
zone of Corticospinal
- Spasticity: Legs > Arms
- Reflections of the sinew: Energetic
- Reflection of Planting: Normal or upgoing
- Samples of Pseudobulbar: Specially with
beginning of the disease > 45 years
- Symmetrical
- No change more under the neuron of the motor
- Sensorial: Normal
- Disfunción of lóobulo frontal: Smooth
- Progression
- Gradual
- Slow
- > 3 years; Up to 3 decades
- Function of vejiga: Normal until late
inside disease
- Laboratory
- Magnetic stimulus: The cortical absentee or
prolonged motor evoked latencies
- MRI: Focal Atrophy of the convolución of
the precentral brain
- Exploration of the DOMESTIC ANIMAL: Reduced
Consumición of the glucose in the pericentral region
- Central times of the conduction of the motor:
Prolonged
- Normal: Serum; Csf; EMG;
Projection of image of the spinal marrow; Serum CK;
Protein of the CSF
- Association of the disease: Cnacer of the
chest
- Pathology
- Zone of Corticospinal: Loss of Axonal
- Normal: Previous cells of Betz ± of the cells of the horn
- Differentiated Diagnosis
- Structural disorders: Spinal; Double
bottle of the hole; Hidrocefalia
- Hereditary spinal disorders
- Infections
PLS: Recesivo, youthful beginning 9
l
Alsin; Chromosome
2q33; Recesivo
- Mutations of gene
- PLS: Canceladura in exon 9
- ALS2: Mutation in another region of gene of
alsin
- Espástico Paraparesis of Familial, infantile
beginning
- Clinical
- Beginning: Childhood
- Spasticity: Bulbar; Extremities
- Watch the paresis
- Normal: Cognition; Sensation
- Laboratory
- Central times of the conduction of the motor:
Retrasado or Unrecordable
- EMG: None denervation
Patient information
Return to the index from polyneuropathy
References
1. Spine 1997;22:486-492
2, J Neurol Sci 1999;170:5-10, JNNP
2001;71:615-620
3, J Neurol 1999;246:1069-1074
4, Clin Neurol Neurosurg 2000;102:109-112
5, JNNP 2000;69:257-261
6, J Neurol 2000;247:654-655
7, Act Neurol Scand 1997;96:14-21
8, Neurology 1999;53:852-855, PNAS
2001;98:6945-6950
9. Genetics 2001;29:160-165, Neuropediatrics
1995;26:313-319 Of the Nature
10, Act Neurol Scand 2003;107:215.220
11, J Neurol Sci 1994;107:153-161, Act Neurol
Scand 1992;85-397-400
8/15/2005 sitemap | index